Subcortical ischemic vascular disease is a disease becomes the leading cause of small to multiple subcortical lacunar infarcts and white matter lesions of the brain damage of ischemic cerebrovascular disease, is caused by vascular cognitive damage to the most common SIVD can cause gait disorders such as Parkinson-like gait, ataxia of gait, ataxia, or no incentive for the frequent falls, a study shows that gait abnormalities may be early signs of vascular This will lead to gait impairment SIVD pathology, type of gait disorder, gait disorder and the relationship between cognitive impairment, gait disorder analysis and evaluation, and treatment were
Subcortical ischemic cerebrovascular disease (with ischemic vascular subcortical, SIVD) is a group to become the main causes, small in subcortical multiple lacunar infarction and cerebral white matter lesions as main brain damage of ischemic cerebrovascular disease, is a cause of vascular cognitive impairment (vascular cognitive resulted VCI), the most common SIVD can cause gait obstacles, such as Parkinson's appearance, all lose tonal gait gait, walking instability or without apparent cause of frequent fall, a study showed that abnormal gait might be early vascular This paper will SIVD gait obstacles in pathological mechanism, gait types, gait obstacles and cognitive impairment of the relationship, gait obstacles analysis and evaluation, Request terminology translation correct statement is smooth, grammar,
dominant-negative版本的表达两个营和CAAT磷酸腺苷反应要素结合蛋白质/ enhancer-binding蛋白质堵住了葡萄糖响应的地区近端”的态度。
This study investigated the effects of Na2SO3on the fat metabolism in human normal diploid HL-7702 (referred as L-02) After 24 h and 48 h, treatment with different concentrations ofNa2SO3, the intra and extra-hepatocellular triglyceride (TG) contents of L-02 were determined usingchemical-enzymatic The contents of very low-density lipoprotein (VLDL) and apolipoproteinB100 (apoB100) in the culture supernatants were determined using enzyme-linked immunosorbentassay (ELISA) Western blot was applied to detect the expressions of fatty acid oxidation and fat synthesisrelated proteins, VLDL assembly and secretion in L-02 本研究在人正常二倍体HL-7702的na2so3on脂肪代谢的影响（称为肝细胞L-02）。后24 h和48 h，用不同浓度ofna2so3处理，内、外肝细胞甘油三酯（TG）含量测定采用化学酶法对L-02。极低密度脂蛋白的含量（VLDL）和apolipoproteinb100（B100）培养上清中均采用酶联免疫吸附试验（ELISA）测定。Western blot检测脂肪酸氧化和脂肪synthesisrelated蛋白的表达，在L-02细胞VLDL的合成与分泌。
这项研究调查了Na2SO3在人体正常二倍体 HL-7702（简称L-02）肝细胞的脂肪代谢作用。在24小时、48小时后，用不同浓度的Na2SO3处理，肝内外细胞……关注进口车市
一个对老鼠繁殖和早期胚胎发展的研究：把药物剂量分成三级：100,300和1000毫克/每天每千克的剂量，在配对前28天开始对公鼠开始注射药物直到公鼠发情位置；而对母鼠，则在配对前14天直到怀孕的第7天注射药物。测试结果是， 剂量在200毫克的时候公鼠的生育率下降，精子活力完全或部分缺失，紊乱的发情周期还有异形精子的比率增大等问题上几乎是人类的11倍。药物对精子的影响可以持续到停止注射药物后9周。
Sensitized Mast Cells Migrate Toward the Agen: A Response Regulated by p38 Mitogen-Activated Protein Kinase and Rho-Associated Coiled-Coil-Forming Protein K致敏（或：敏化）肥大细胞趋向抗原：由p38促分裂原活化蛋白激酶（MAPK）与Rho相关超螺旋蛋白激酶调控的反应。。。供参
都是费事的事情，为什么不自己去找或者花钱买吧
Sensitized Mast Cells Migrate Toward the Agen: A Response Regulated by p38 Mitogen-Activated Protein Kinase and Rho-Associated Coiled-Coil-Forming Protein K致敏（或：敏化）肥大细胞趋向抗原：由p38促分裂原活化蛋白激酶（MAPK）与Rho相关超螺旋蛋白激酶调控的反应。。。供参
Subcortical ischemic cerebrovascular disease is a group to become the main causes, small in subcortical multiple lacunar infarction and cerebral white matter lesions as main brain damage of ischemic cerebrovascular disease, is the most common vascular cognitive impairment of SIVD can cause gait obstacles, such as Parkinson's appearance, all lose tonal gait gait, walking instability or without apparent cause of frequent fall, a study showed that abnormal gait might be early vascular This paper will SIVD gait obstacles in pathological mechanism, gait types, gait obstacles and cognitive impairment of the relationship, gait obstacles analysis and evaluation,
Subcortical ischemic cerebrovascular disease (with ischemic vascular subcortical, SIVD) is a group to become the main causes, small in subcortical multiple lacunar infarction and cerebral white matter lesions as main brain damage of ischemic cerebrovascular disease, is a cause of vascular cognitive impairment (vascular cognitive resulted VCI), the most common SIVD can cause gait obstacles, such as Parkinson's appearance, all lose tonal gait gait, walking instability or without apparent cause of frequent fall, a study showed that abnormal gait might be early vascular This paper will SIVD gait obstacles in pathological mechanism, gait types, gait obstacles and cognitive impairment of the relationship, gait obstacles analysis and evaluation,
Subcortical ischemic cerebrovascular disease (with ischemic vascular subcortical, SIVD) is a group to become the main causes, small in subcortical multiple lacunar infarction and cerebral white matter lesions as main brain damage of ischemic cerebrovascular disease, is a cause of vascular cognitive impairment (vascular cognitive resulted VCI), the most common SIVD can cause gait obstacles, such as Parkinson's appearance, all lose tonal gait gait, walking instability or without apparent cause of frequent fall, a study showed that abnormal gait might be early vascular This paper will SIVD gait obstacles in pathological mechanism, gait types, gait obstacles and cognitive impairment of the relationship, gait obstacles analysis and evaluation, Request terminology translation correct statement is smooth, grammar,
Subcortical ischemic cerebrovascular disease (with ischemic vascular subcortical, SIVD) is a group to become the main causes, small in subcortical multiple lacunar infarction and cerebral white matter lesions as main brain damage of ischemic cerebrovascular disease, is a cause of vascular cognitive impairment (vascular cognitive resulted VCI), the most common SIVD can cause gait obstacles, such as Parkinson's appearance, all lose tonal gait gait, walking instability or without apparent cause of frequent fall, a study showed that abnormal gait might be early vascular This paper will SIVD gait obstacles in pathological mechanism, gait types, gait obstacles and cognitive impairment of the relationship, gait obstacles analysis and evaluation,
一个对老鼠繁殖和早期胚胎发展的研究：把药物剂量分成三级：100,300和1000毫克/每天每千克的剂量，在配对前28天开始对公鼠开始注射药物直到公鼠发情位置；而对母鼠，则在配对前14天直到怀孕的第7天注射药物。测试结果是， 剂量在200毫克的时候公鼠的生育率下降，精子活力完全或部分缺失，紊乱的发情周期还有异形精子的比率增大等问题上几乎是人类的11倍。药物对精子的影响可以持续到停止注射药物后9周。
dominant-negative版本的表达两个营和CAAT磷酸腺苷反应要素结合蛋白质/ enhancer-binding蛋白质堵住了葡萄糖响应的地区近端”的态度。
Subcortical ischemic vascular disease is a disease becomes the leading cause of small to multiple subcortical lacunar infarcts and white matter lesions of the brain damage of ischemic cerebrovascular disease, is caused by vascular cognitive damage to the most common SIVD can cause gait disorders such as Parkinson-like gait, ataxia of gait, ataxia, or no incentive for the frequent falls, a study shows that gait abnormalities may be early signs of vascular This will lead to gait impairment SIVD pathology, type of gait disorder, gait disorder and the relationship between cognitive impairment, gait disorder analysis and evaluation, and treatment were
Sensitized Mast Cells Migrate Toward the Agen: A Response Regulated by p38 Mitogen-Activated Protein Kinase and Rho-Associated Coiled-Coil-Forming Protein K致敏（或：敏化）肥大细胞趋向抗原：由p38促分裂原活化蛋白激酶（MAPK）与Rho相关超螺旋蛋白激酶调控的反应。。。供参
这项研究调查了Na2SO3在人体正常二倍体 HL-7702（简称L-02）肝细胞的脂肪代谢作用。在24小时、48小时后，用不同浓度的Na2SO3处理，肝内外细胞……关注进口车市
This study investigated the effects of Na2SO3on the fat metabolism in human normal diploid HL-7702 (referred as L-02) After 24 h and 48 h, treatment with different concentrations ofNa2SO3, the intra and extra-hepatocellular triglyceride (TG) contents of L-02 were determined usingchemical-enzymatic The contents of very low-density lipoprotein (VLDL) and apolipoproteinB100 (apoB100) in the culture supernatants were determined using enzyme-linked immunosorbentassay (ELISA) Western blot was applied to detect the expressions of fatty acid oxidation and fat synthesisrelated proteins, VLDL assembly and secretion in L-02 本研究在人正常二倍体HL-7702的na2so3on脂肪代谢的影响（称为肝细胞L-02）。后24 h和48 h，用不同浓度ofna2so3处理，内、外肝细胞甘油三酯（TG）含量测定采用化学酶法对L-02。极低密度脂蛋白的含量（VLDL）和apolipoproteinb100（B100）培养上清中均采用酶联免疫吸附试验（ELISA）测定。Western blot检测脂肪酸氧化和脂肪synthesisrelated蛋白的表达，在L-02细胞VLDL的合成与分泌。
Expression of dominant-negative versions of both cAMP response element-binding protein and CAAT/enhancer-binding protein blocked the glucose response of the proximal region in a dose-dependent mannerdominant-negative版本的表达两个营和CAAT磷酸腺苷反应要素结合蛋白质/ enhancer-binding蛋白质堵住了葡萄糖响应的地区近端”的态度